Uric acid is the final product of purine metabolism in humans. Asymptomatic hyperuricemia is frequently detected through biochemical screening. The major causes of hyperuricemia are increased purine synthesis, inherited metabolic disorder, excess dietary purine intake, increased nucleic acid turnover, malignancy, cytotoxic drugs, and decreased excretion due to chronic renal failure or increased renal reabsorption. Long term follow-up of these patients is undertaken because many are at risk of renal disease that may develop; few of these patients ever develop the clinical syndrome of gout.
|Adult male||:|| 150-450 μmol/L
|Adult female||:|| 150-350 μmol/L
|Less than 1 year||:|| 80-370 μmol/L
|1-12 years||:||120-300 μmol/L|
SST (gold top)