The majority of body calcium is bone-associated and circulating levels reflect bone turnover and disease. Circulating calcium is under the control of parathyroid hormone. The parathyroid gland mediates increased serum calcium as a function of PTH production. Primary hyperparathyroidism reflects adenoma/carcinoma or hyperplastic syndromes, leading to excess serum calcium as a function of bone mineralisation and increased renal reabsorption/intestinal uptake. Secondary forms typically reflect renal failure.
|Less than 1 month||:|| 2.0-2.7 mmoll/L
|1 month-16 years||:|| 2.2-2.7 mmol/L
|Greater than 16 years||:||2.15-2.60 mmol/L|
SST (gold top)